Search results for "endocrine disruptors"

showing 10 items of 52 documents

Effect of methylparaben in Artemia franciscana.

2017

In this study, the toxicity of methylparaben (MeP) an emerging contaminant, was analysed in the sexual species Artemia franciscana, due to its presence in coastal areas and marine saltworks in the Mediterranean region. The acute toxicity (24 h-LC50) of MeP in nauplii was tested and its chronic effect (9 days) evaluated by measuring survival and growth under two sublethal concentrations (0.0085 and 0.017 mg/L). Also, the effect on several key enzymes involved in: antioxidant defences (catalase (CAT) and gluthathion-S-transferase (GST)), neural activity (cholinesterase (ChE)) and xenobiotic biotransformation (carboxylesterase (CbE), was assessed after 48 h under sublethal exposure. The result…

0106 biological sciencesAntioxidantPhysiologyHealth Toxicology and Mutagenesismedicine.medical_treatmentAquaculture010501 environmental sciencesEndocrine DisruptorsToxicologymedicine.disease_cause01 natural sciencesBiochemistryToxicologyCarboxylesterasechemistry.chemical_compoundMorphogenesisFood scienceToxicity Tests ChronicBiotransformationbiologyMethylparabenArtemia franciscanaGeneral MedicineBiodiversityCatalaseToxicokineticsCatalaseLarvaToxicityParabensArthropod ProteinsLethal Dose 50medicineToxicity Tests AcuteAnimals0105 earth and related environmental sciencesCholinesteraseToxicity010604 marine biology & hydrobiologyPreservatives PharmaceuticalCell BiologySurvival AnalysisAcute toxicityOxidative Stresschemistrybiology.proteinCarcinogensArtemiaXenobioticOxidative stressBiomarkersWater Pollutants ChemicalComparative biochemistry and physiology. Toxicologypharmacology : CBP
researchProduct

Early endocrine disruptors exposure acts on 3T3-L1 differentiation and endocrine activity

2017

International audience; Introduction: Data from last years suggested that early exposure to endocrine disruptors (EDs) can predispose newborns to endocrine dysfunction of adipocytes, obesity, and associated disorders. The implication of EDs at low doses on adipocyte development has been poorly investigated. For instance, vinclozolin (V) is a dicarboximide fungicide widely used in agriculture since the 90's, alone or in mixture with genistein (G), an isoflavonoid from Leguminosae. This study aims to identify the effect of vinclozolin alone or with genistein, on adipose tissue properties using cell culture.Methods: In steroid-free conditions, 3T3-L1 pre-adipocytes were induced to differentiat…

0301 basic medicineLeptin[ SDV.TOX.ECO ] Life Sciences [q-bio]/Toxicology/Ecotoxicologytissu adipeuxPharmaceutical ScienceGenisteinAdipose tissueoestradiolsourisTriglyceridechemistry.chemical_compound0302 clinical medicineAdipocyteratVinclozolinlcsh:QH301-705.5Original Researchperturbateur endocrinienlcsh:R5-920LeptinGeneral MedicineGenistein[ SDV.TOX.TCA ] Life Sciences [q-bio]/Toxicology/Toxicology and food chainobésité030220 oncology & carcinogenesisAlimentation et NutritionEndocrinologie et métabolismeleptine[SDV.TOX.ECO]Life Sciences [q-bio]/Toxicology/Ecotoxicologylcsh:Medicine (General)medicine.medical_specialtyAdipose tissue[SDV.TOX.TCA]Life Sciences [q-bio]/Toxicology/Toxicology and food chainBiologyadipocyteGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciencesInternal medicinemedicineFood and NutritionEndocrine systemEndocrine disruptorsEndocrinology and metabolism3T3-L1adipose tissue;endocrine disruptors;genistein;triglyceride;leptin;vinclozolin;promotes adipocyte differentiation;adipose-tissue development;rat;adipocytes;mice;obesity;expression;estradiol030104 developmental biologyEndocrinologylcsh:Biology (General)chemistryVinclozolinCell culture
researchProduct

Prenatal exposure to mixtures of xenoestrogens and genome-wide DNA methylation in human placenta

2015

BACKGROUND: In utero exposure to xenostrogens may modify the epigenome. We explored the association of prenatal exposure to mixtures of xenoestrogens and genome-wide placental DNA methylation. MATERIALS & METHODS: Sex-specific associations between methylation changes in placental DNA by doubling the concentration of TEXB-alpha exposure were evaluated by robust multiple linear regression. Two CpG sites were selected for validation and replication in additional male born placentas. RESULTS: No significant associations were found, although the top significant CpGs in boys were located in the LRPAP1, HAGH, PPARGC1B, KCNQ1 and KCNQ1DN genes, previously associated to birth weight, Type 2 diabetes…

0301 basic medicineMaleCancer Researchmedicine.medical_specialtymedicine.medical_treatmentPlacentaADNBiologyEpigenesis GeneticAndrology03 medical and health scienceschemistry.chemical_compoundSex FactorsPregnancyPlacentaInternal medicineGeneticsmedicineBirth WeightHumansPrenatalLDL-Receptor Related Protein-Associated ProteinGeneTEXBEndocrine disruptorsDNA methylationEpigenome xenoestrogensRNA-Binding ProteinsEstrogensMethylationEpigenomeSteroid hormone030104 developmental biologyEndocrinologymedicine.anatomical_structureXenoestrogenCpG sitechemistryPrenatal Exposure Delayed EffectsDNA methylationKCNQ1 Potassium ChannelProgrammingCpG IslandsFemaleThiolester HydrolasesCarrier ProteinsGenome-Wide Association Study
researchProduct

Prenatal exposure to endocrine disrupting chemicals and risk of being born small for gestational age: Pooled analysis of seven European birth cohorts

2018

Background and aims: There is evidence that endocrine disrupting chemicals (EDCs) have developmental effects at environmental concentrations. We investigated whether some EDCs are associated with the adverse birth outcome Small for Gestational Age (SGA). Methods: We used PCB 153, p,p'-DDE, HCB, PFOS and PFOA measured in maternal, cord blood or breast milk samples of 5446 mother-child pairs (subset of 693 for the perfluorinated compounds) from seven European birth cohorts (1997–2012). SGA infants were those with birth weight below the 10th percentile for the norms defined by gestational age, country and infant's sex. We modelled the association between measured or estimated cord serum EDC co…

0301 basic medicineMalemedicine.medical_specialtyBirth weight010501 environmental sciencesBreast milkEndocrine Disruptors01 natural sciencesPooled analysis03 medical and health sciencesSDG 3 - Good Health and Well-beingInterquartile rangePregnancyMedicine[SDV.EE.SANT] Life Sciences [q-bio]/Ecology environment/HealthHumansBiologylcsh:Environmental sciences0105 earth and related environmental sciencesGeneral Environmental Science2. Zero hungerlcsh:GE1-350[SDV.EE.SANT]Life Sciences [q-bio]/Ecology environment/HealthPregnancyMilk Humanbusiness.industryObstetricsSmokingInfant NewbornGestational ageEnvironmental exposureOdds ratioEndocrine disrupting chemicals (EDCs)Infant Low Birth Weightmedicine.diseaseFetal Blood3. Good healthChemistrySmall for gestational age (SGA)030104 developmental biology[SDV.SPEE] Life Sciences [q-bio]/Santé publique et épidémiologieMaternal ExposurePrenatal Exposure Delayed EffectsSmall for gestational age/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingFemale[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologiebusiness
researchProduct

Impairment of learning and memory performances induced by BPA Evidences from the literature of a MoA mediated through an ED

2018

International audience; Many rodent studies and a few non-human primate data report impairments of spatial and non-spatial memory induced by exposure to bisphenol A (BPA), which are associated with neural modifications, particularly in processes involved in synaptic plasticity. BPA-induced alterations involve disruption of the estrogenic pathway as established by reversal of BPA-induced effects with estrogenic receptor antagonist or by interference of BPA with administered estradiol in ovariectomized animals. Sex differences in hormonal impregnation during critical periods of development and their influence on maturation of learning and memory processes may explain the sexual dimorphism obs…

0301 basic medicineNervous systemNervous systemendocrine systemmedicine.drug_classEndocrine disruptionBiologyEndocrine DisruptorsBiochemistryLearning and memory03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologyMESH: PhenolsBisphenol APhenolsMemorymedicineMESH: Benzhydryl CompoundsAnimalsHumansBenzhydryl compoundsMESH: MemoryBenzhydryl CompoundsMode of actionMolecular BiologyBehavior Animalurogenital systemBrainCognitionEnvironmental exposureEnvironmental ExposureReceptor antagonistMESH: Endocrine Disruptors030104 developmental biologymedicine.anatomical_structurechemistrySynaptic plasticity[SDV.SPEE]Life Sciences [q-bio]/Santé publique et épidémiologieSignal transductionNeuroscience030217 neurology & neurosurgeryhormones hormone substitutes and hormone antagonists
researchProduct

An in vitro investigation on the cytotoxic and nuclear receptor transcriptional activity of the mycotoxins fumonisin B1 and beauvericin.

2016

Fumonisin B1 (FB1) and beauvericin (BEA) are secondary metabolites of filamentous fungi, which under appropriate temperature and humidity conditions may develop on various foods and feeds. To date few studies have been performed to evaluate the toxicological and endocrine disrupting effects of FB1 and BEA. The present study makes use of various in vitro bioassays including; oestrogen, androgen, progestagen and glucocorticoid reporter gene assays (RGAs) for the study of nuclear receptor transcriptional activity, the thiazolyl blue tetrazolium bromide (MTT) assay to monitor cytotoxicity and high content analysis (HCA) for the detection of pre-lethal toxicity in the RGA and Caco-2 human colon …

0301 basic medicineTranscription GeneticCell SurvivalBiologyAdenocarcinomaEndocrine DisruptorsToxicologyFumonisins03 medical and health scienceschemistry.chemical_compound0404 agricultural biotechnologyGlucocorticoid receptorReceptors GlucocorticoidGenes ReporterDepsipeptidesmedicineHumansCytotoxicityReceptorCell NucleusFumonisin B1Dose-Response Relationship Drug04 agricultural and veterinary sciencesGeneral Medicine040401 food scienceBeauvericin030104 developmental biologychemistryNuclear receptorBiochemistryReceptors AndrogenToxicityColonic NeoplasmsCaco-2 CellsReceptors ProgesteroneGlucocorticoidmedicine.drugToxicology letters
researchProduct

Bisphenolic compounds alter gene expression in MCF-7 cells through interaction with estrogen receptor α

2020

Plasticizers released from microplastic are increasingly viewed with concern. While adverse health effects induced by bisphenol A and its analogues on marine animals are well documented in the literature, the endocrine potential of bisphenolic compounds on human health remains elusive. We applied next generation sequencing (NGS) with the estrogen receptor α (ERα) positive human breast cancer cell line MCF-7 treated with 17-β-estradiol (E2), bisphenol A (BPA), bisphenol B (BPB), bisphenol Z (BPZ) and tetramethyl bisphenol A (4MeBPA). We used molecular docking, microscale thermophoresis, ERα activation assay, and cell cycle experiments on MCF-7 and ERα overexpressing HEK293 cells to verify th…

0301 basic medicineendocrine systemBisphenolDown-RegulationGene ExpressionEstrogen receptorBreast NeoplasmsEndocrine DisruptorsToxicologyCell Line03 medical and health sciences0302 clinical medicinePhenolsPlasticizersBCAS3Cell Line TumorHumansBenzhydryl CompoundsCell ProliferationInsulin-like growth factor 1 receptorPharmacologyEstradiolChemistryCell growthEstrogen Receptor alphaEstrogensCell cycleUp-RegulationHEK293 Cells030104 developmental biologyPRKCDMCF-7030220 oncology & carcinogenesisMCF-7 CellsCancer researchFemalehormones hormone substitutes and hormone antagonistsSignal TransductionToxicology and Applied Pharmacology
researchProduct

Regulatory identification of BPA as an endocrine disruptor : Context and methodology

2018

International audience; BPA is one of the most investigated substances for its endocrine disruptor (ED) properties and it is at the same time in the center of many ED-related controversies, the analysis on how BPA fits to the regulatory identification as an ED is a challenge in terms of methodology. It is also a great opportunity to test the regulatory framework with a uniquely data-rich substance and learn valuable lessons for future cases. From this extensive database, it was considered important to engage in a detailed analysis so as to provide specific and strong evidences of ED while reflecting accurately the complexity of the response as well the multiplicity of adverse effects. An ap…

0301 basic medicineendocrine systemBrain developmentpertubateur endocrinienEndocrine disruptionContext (language use)Computational biologyMESH: Social Control Formal010501 environmental sciencesBiologyEndocrine Disruptors01 natural sciencesBiochemistryMammary gland developmentbisphenol A;endocrine disruption;ED;SVHC;substance of very high concern;REACh03 medical and health sciencesEndocrinologyBisphenol AMESH: PhenolsPhenolsSubstance of very high concernMESH: Benzhydryl CompoundsFood and Nutritionbisphénol aAnimalsHumansBenzhydryl CompoundsMolecular BiologyComputingMilieux_MISCELLANEOUSSVHC0105 earth and related environmental sciencesEndocrinology and metabolismurogenital systemsubstance toxiqueED3. Good healthSocial Control FormalMESH: Endocrine Disruptors030104 developmental biologyEndocrine disruptor[SDV.TOX]Life Sciences [q-bio]/ToxicologyAlimentation et NutritionEndocrinologie et métabolismeREACHIdentification (biology)
researchProduct

Incidence of thyroid cancer in Italian contaminated sites

2020

Some human literature suggests a possible role of endocrine disruptors (EDs) exposure in thyroid cancer (TC) development. We investigated TC incidence in selected Italian National Priority Contaminated Sites (NPCS) with documented presence of EDs considered thyroid carcinogens. Adjusted Standardized Incidence Ratios (SIRs), with their 90% confidence intervals, were computed by gender, and age-specific groups (aged 15–39 years, and 40 years or over) for each NPCS in the period 2006 to 2013. In the age group of 15–39 years, a significant excess of TC risk was found in two NPCSs in males

AdultMaleAdolescentHealth Toxicology and Mutagenesislcsh:Medicine010501 environmental sciences01 natural sciencesArticleThyroid cancer03 medical and health sciencesYoung Adult0302 clinical medicinemedicineHumansThyroid NeoplasmsRisk factorAdverse effectThyroid cancer0105 earth and related environmental sciencesbusiness.industryIncidence (epidemiology)IncidenceThyroidlcsh:RPublic Health Environmental and Occupational HealthEcological studyEnvironmental exposureEnvironmental Exposuremedicine.diseaseConfidence intervalmedicine.anatomical_structureendocrine disruptorsEndocrine disruptorItaly030220 oncology & carcinogenesisFemalebusinessEnvironmental PollutionDemographyHuman
researchProduct

Risk analysis of main mycotoxins occurring in food for children: An overview

2015

Mycotoxins are secondary metabolites produced by fungi contaminating the food chain that are toxic to animals and humans. Children up to 12 years old are recognized as a potentially vulnerable subgroup with respect to consumption of these contaminants. Apart from having a higher exposure per kg body weight, they have a different physiology from that of adults. Therefore they may be more sensitive to neurotoxic, endocrine and immunological effects. For these reasons, a specific and up-to-date risk analysis for this category is of great interest. In this review, an accurate analysis of the main mycotoxins occurring in food intended for children (deoxynivalenol, aflatoxins, ochratoxins, patuli…

AflatoxinRisk analysisFood ContaminationEndocrine DisruptorsBiologyToxicologyRisk AssessmentOchratoxinsBaby foodPatulinFood chainchemistry.chemical_compoundAflatoxinAnimalsHumansChildMycotoxinChildrenBaby foodFumonisinInfantfood and beveragesGeneral MedicineMycotoxinsDeoxynivalenolCarcinogens EnvironmentalToxicokineticsOchratoxinPatulinRisk analysis (engineering)chemistryChild PreschoolInfant FoodRisk assessmentFood ScienceFood contaminantFood and Chemical Toxicology
researchProduct